Christoph Mulert

This study examines if glycine improves negative symptoms in the ketamine model of schizophrenia by modulating auditory evoked gamma-band responses.

The paper identifies pyramidal neurons in the prefrontal cortex as key elements causing abnormal circuit function in a mouse model of mental illness and shows that microglia inhibition rescues these deficits.

This study localizes the auditory evoked gamma band response (aeGBR) sources and examines their connectivity using MEG, finding increased dACC activity and connectivity during cognitively demanding tasks.