Author

Rossana Aprigliano

University of ZurichORCID 0000-0001-8537-122XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:ChIP-seq· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:statistical model· 1

Frequent Co-Authors

Marcin Roganowicz· 2 shared Raffaella SantoroiD· 2 shared Cristiana BersaglieriiD· 1 shared Dominik Bär· 1 shared Juliana Bizzarro· 1 shared Karolina Pietrzak· 1 shared Luigi LerraiD· 1 shared Rodrigo Peña‐Hernández· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersMolecular Biology

Marcin Roganowicz

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2 papers

Recent·Most cited

Molecular BiologyLife Science AlliancePeer-reviewedApr 25, 2023

BAZ2A-RNA mediated association with TOP2A and KDM1A represses gene expression in prostate cancer

Marcin Roganowicz, Dominik Bär, Cristiana Bersaglieri…·Raffaella Santoro

The paper identifies a novel RNA-based mechanism in prostate cancer where BAZ2A interacts with TOP2A and KDM1A to repress gene expression, distinct from its role in rRNA gene silencing.

9 citations

human

𝕏 PostBluesky

Molecular BiologyEMBO ReportsPeer-reviewedAug 17, 2021

BAZ2A association with H3K14ac is required for the transition of prostate cancer cells into a cancer stem-like state

Rodrigo Peña‐Hernández, Rossana Aprigliano, Sandra C. Frommel…·Raffaella Santoro

This study shows BAZ2A's role in prostate cancer stem-like state transition through its interaction with H3K14ac and repression of tumor suppressor genes.

35 citationstop 5%

humanDNA methylation

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