Gábor Juhász

The paper identifies multiple SNARE-dependent pathways driving endosome maturation in Drosophila nephrocytes, revealing a network of parallel fusion routes rather than a single linear pathway.

This study identifies Hip14 and Patsas as rate-limiting factors in lysosome formation and fusion, linking defective palmitoylation to Huntington's disease-like symptoms in Drosophila.

The paper identifies Rab7-Epg5 and Rab39-ema modules that coordinate autophagosome positioning toward the ncMTOC for efficient lysosomal fusion in Drosophila fat cells.

The paper identifies ubiquitination as a key trigger for crinophagic degradation of secretory granules in Drosophila, mediated by Cnot4 and Usp7.

The paper shows that autophagy degrades Su(var)2-10/PIAS to activate Stat92E in Drosophila glial cells during injury, enhancing glial reactivity and innate immune responses.