Maximilian Schindler

Glomage enables high-throughput analysis of glomeruli in zebrafish and mouse models, combining morphological and RNA profiling to study podocyte changes and drug responses.

This study identifies MAGI2 as a conserved biomarker for podocyte injury, showing its downregulation in glomerular diseases and differential expression in primary vs secondary FSGS.

This study introduces Nifurpirinol as a more efficient prodrug for inducing FSGS-like glomerular injury in zebrafish larvae, suitable for high-content drug screening.

This paper demonstrates that synthetic CTNS mRNA therapy can restore cystinosin function and reduce cystine accumulation in kidney cells and zebrafish models of cystinosis.