Author

Marcos Eliezeck

Universidade Federal de Minas GeraisORCID 0000-0001-8942-6822Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Physiology· 2

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:rat· 1 Approach:statistical model· 1

Frequent Co-Authors

B. d. L. SanchesiD· 2 shared Lucas Rodrigues‐RibeiroiD· 2 shared Maria José Campagnole‐SantosiD· 2 shared Robson A.S. SantosiD· 2 shared Sérgio ScalzoiD· 2 shared Sílvia GuatimosimiD· 2 shared Thiago Verano‐BragaiD· 2 shared Adelson Héric Alves MonteiroiD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersPhysiology

B. d. L. SanchesORCID

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2 papers

Recent·Most cited

PhysiologyCirculation ResearchPeer-reviewedNov 12, 2025

Identification and characterization of alamandine-(1-5), a new component of the Renin-Angiotensin System with unique properties

Robson A.S. Santos, Melissa Tainan Silva Dias, Amanda de Sá Martins de Bessa…·Maria José Campagnole‐Santos

This study identifies and characterizes alamandine-(1-5), a new RAS component with unique biological activities and increased plasma concentration in nephropathic patients.

10 citationstop 3%

humanmouse

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PhysiologybioRxivPreprintMar 27, 2024

Evidence that the monoamine oxidase B (MAO-B) plays a central role in the inotropic dysfunction induced by genetic deletion of the Mas-related-G protein-coupled receptor D (MrgD) in mice

Lucas Rodrigues‐Ribeiro, Júlia Rezende-Ribeiro, Sérgio Scalzo…·Thiago Verano‐Braga

This study identifies MAO-B hyperactivation as a key contributor to cardiac dysfunction in MrgD-KO mice, showing that MAO-B inhibition reverses contractile defects.

DOI 10.1101/2024.03.27.586916

mouseproteomics

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