Author

Bruno Sanches

Universidade Federal de Minas GeraisORCID 0000-0002-9029-609XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:mouse· 3 Assay:proteomics· 3 Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:rat· 1 Approach:statistical model· 1

Frequent Co-Authors

Sílvia GuatimosimiD· 3 shared Thiago Verano‐BragaiD· 3 shared Júlia Rezende RibeiroiD· 2 shared Lucas Rodrigues‐RibeiroiD· 2 shared Marcos EliezeckiD· 2 shared Maria José Campagnole‐SantosiD· 2 shared Maria Luiza Dias-PintoiD· 2 shared Robson A.S. SantosiD· 2 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersPhysiology

Sílvia GuatimosimORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

PhysiologybioRxivPreprintOct 9, 2026

Oral administration of Angiotensin-(1-7) included in hydroxypropyl β-cyclodextrin attenuates cardiac hypertrophy and inflammation in a mouse model of Takotsubo cardiomyopathy

Maria Luiza Dias-Pinto, Bruno Sanches, Júlia Rezende Ribeiro…·Thiago Verano‐Braga

DOI 10.64898/2026.10.01.756123

mouseproteomics

PhysiologyCirculation ResearchPeer-reviewedNov 12, 2025

Identification and characterization of alamandine-(1-5), a new component of the Renin-Angiotensin System with unique properties

Robson A.S. Santos, Melissa Tainan Silva Dias, Amanda de Sá Martins de Bessa…·Maria José Campagnole‐Santos

This study identifies and characterizes alamandine-(1-5), a new RAS component with unique biological activities and increased plasma concentration in nephropathic patients.

10 citationstop 3%

PhysiologybioRxivPreprintMar 27, 2024

Evidence that the monoamine oxidase B (MAO-B) plays a central role in the inotropic dysfunction induced by genetic deletion of the Mas-related-G protein-coupled receptor D (MrgD) in mice

Lucas Rodrigues‐Ribeiro, Júlia Rezende-Ribeiro, Sérgio Scalzo…·Thiago Verano‐Braga

This study identifies MAO-B hyperactivation as a key contributor to cardiac dysfunction in MrgD-KO mice, showing that MAO-B inhibition reverses contractile defects.

DOI 10.1101/2024.03.27.586916

mouseproteomics