Hailong Dong

The paper shows that microglial process dynamics shield GABAergic inputs to enhance neuronal activity after anesthesia in mice.

This study shows spinal microglia amplify neuronal activity in inflammatory pain via the P2Y12 receptor, suggesting a therapeutic target for pain management.

This study shows that optogenetic activation of spinal microglia in mice induces chronic pain through IL-1 signaling and increased neuronal activity.

This study shows that dysfunction of VTA GABA neurons leads to mania-like behaviors, including increased wakefulness and reduced sleep, which can be reversed by valproate.

This study shows that neuronal activity in awake mice influences microglial process surveillance via norepinephrine signaling, highlighting the need for awake imaging in studying microglia-neuron interactions.