Author

Ashok Daniel Prabakaran

Cincinnati Children's Hospital Medical CenterORCID 0000-0001-9053-2673Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Physiology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:ChIP-seq· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Hannah LatimeriD· 2 shared Hima Bindu DurumutlaiD· 2 shared Kevin McFarland· 2 shared Mattia QuattrocelliiD· 2 shared Olukunle AkinborewaiD· 2 shared Akanksha Rajput· 1 shared April M. HalleriD· 1 shared Bahram Namjou‐Khales· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersPhysiology

Hannah LatimerORCID

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2 papers

Recent·Most cited

PhysiologyScience AdvancesPeer-reviewedJul 9, 2025

The human genetic variant rs6190 unveils Foxc1 and Arid5a as novel pro-metabolic targets of the glucocorticoid receptor in muscle.

Ashok Daniel Prabakaran, Hyun-Jy Chung, Kevin McFarland…·Mattia Quattrocelli

This study identifies Foxc1 and Arid5a as novel pro-metabolic targets of the glucocorticoid receptor in muscle, using a human SNP rs6190 in mice and human datasets.

5 citations

mousebulk RNA-seq

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PhysiologyJournal of Clinical InvestigationPeer-reviewedJul 1, 2025

The human glucocorticoid receptor variant rs6190 promotes blood cholesterol and atherosclerosis

Hima Bindu Durumutla, April M. Haller, Greta Noble…·Mattia Quattrocelli

This study identifies a human GR variant, rs6190, that increases cholesterol and atherosclerosis risk by enhancing GR activity in mice and human cells.

7 citationstop 3%

humanmouse

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