Author

Marie Fernandes

Centre de Recherches en Cancérologie de LilleORCID 0000-0001-9150-2804Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:clustering· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

David TulasneiD· 2 shared Jean‐Pascal MenebooiD· 2 shared Marie‐José TruongiD· 2 shared Martin FigeaciD· 2 shared Agathe LaratteiD· 1 shared Alexis Benjamin CortotiD· 1 shared Andréa ParadisiiD· 1 shared Anne Chotteau‐LelièvreiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCancer Biology

David TulasneORCID

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2 papers

Recent·Most cited

Oncogenic mutations convert MET from a pro-apoptotic tumor suppressor to an oncogenic driver

Rémi Tellier, Marie Fernandes, Audrey Vinchent, Sonia Paget, Agathe Laratte, Céline Vuillier, Elisabeth Werkmeister, Céline Villenet, Jean‐Pascal Meneboo, Anne Chotteau‐Lelièvre, Eric Wasielewski, Zoulika Kherrouche, Marie‐José Truong, Clotilde Descarpentries, Luca Grumolato, , David Tulasne

The paper shows that MET exon 14 skipping mutations eliminate the pro-apoptotic function of MET, converting it from a tumor suppressor to an oncogenic driver in lung adenocarcinomas.

bioRxiv · Nov 10, 2025 · PreprintCancer Biology

Transcriptional program-based deciphering of the MET exon 14 skipping regulation network

Marie‐José Truong, Geoffrey Pawlak, Jean‐Pascal Meneboo, Shéhérazade Sebda, Marie Fernandes, Martin Figeac, Mohamed Elati, David Tulasne

The paper identifies a regulatory network involving ETS1, FOSL1, and SMAD3 that mediates MET exon 14 skipping in lung cancer, showing their role in RAS-ERK pathway activation and potential as therapeutic targets.

Cell Death and Disease · Nov 3, 2025Peer-reviewedCancer Biology