Author

Amy L. Pashler

Brighton and Sussex Medical SchoolORCID 0000-0001-9241-8663Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:database / resource· 1 Research Focus:differential expression· 1 Organism:Drosophila· 1 Research Focus:gene regulatory network inference· 1 Approach:statistical model· 1

Frequent Co-Authors

Benjamin P. TowleriD· 2 shared Hope J. HaimeiD· 2 shared Sarah Faith NewburyiD· 2 shared Cecília M. ArraianoiD· 1 shared Christopher Iain JonesiD· 1 shared Katarzyna M. Przybyl· 1 shared Rute Gonçalves MatosiD· 1 shared Sandra C. ViegasiD· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersMolecular Biology

Benjamin P. TowlerORCID

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2 papers

Recent·Most cited

Molecular BiologyRNAPeer-reviewedJul 15, 2021

Genome-wide analyses of XRN1-sensitive targets in osteosarcoma cells identifies disease-relevant transcripts containing G-rich motifs.

Amy L. Pashler, Benjamin P. Towler, Christopher Iain Jones…·Sarah Faith Newbury

The paper identifies XRN1-sensitive transcripts in osteosarcoma cells and shows they contain G-rich motifs, suggesting XRN1 regulates lncRNAs involved in disease progression.

7 citations

humanbulk RNA-seq

𝕏 PostBluesky

Molecular BiologybioRxivPreprintOct 16, 2019

Dis3L2 regulates cellular proliferation through a PI3-Kinase dependent signalling pathway

Benjamin P. Towler, Amy L. Pashler, Hope J. Haime…·Sarah Faith Newbury

This study identifies Dis3L2 as a regulator of cell proliferation through a PI3-Kinase dependent pathway in both Drosophila and human cells.

1 citation

Drosophilahuman

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