Nipavan Chiamvimonvat

The paper quantifies cellular organization and connexin expression in the porcine sinoatrial node, revealing regional differences in pacemaking function and current source-sink relationships.

This study identifies ceramides as mediators of mitochondrial dysfunction in sinoatrial node cells during heart failure, suggesting they as potential therapeutic targets.

This study investigates KCNJ2 mutations linked to ventricular arrhythmia using computational modeling and functional analysis, revealing mutation-specific structural perturbations and their impact on channel stability.

This study shows chronic AngII signaling disrupts cardiac phosphoinositide homeostasis, leading to cardiac dysfunction, but PTEN inhibition protects by preserving PI(3,4,5)P3 levels and reducing fibrosis.