Author

Fernando Mejías-Navarro

Centro Andaluz de Biología Molecular y Medicina RegenerativaORCID 0000-0001-9543-0676Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetics· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:CRISPR screen· 1 Assay:DNA methylation· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Approach:statistical model· 1 Research Focus:variant calling· 1

Frequent Co-Authors

Pablo HuertasiD· 2 shared Amador Romero-FrancoiD· 1 shared Andrés AguileraiD· 1 shared Angela GalloiD· 1 shared Daniel Gómez-CabelloiD· 1 shared Domenico Alessandro SilvestrisiD· 1 shared Guillermo Rodríguez‐RealiD· 1 shared Judit Domingo‐PrimiD· 1 shared

Activity by Year

2018-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersGenetics

Pablo HuertasORCID

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2 papers

Recent·Most cited

GeneticsNature CommunicationsPeer-reviewedSep 17, 2021

ADAR2-mediated RNA editing of DNA:RNA hybrids is required for DNA double strand break repair

Sonia Jimeno, Rosario Prados‐Carvajal, María Jesús Fernández-Ávila…·Pablo Huertas

The paper shows that ADAR2-mediated RNA editing of DNA:RNA hybrids is essential for DNA double strand break repair by facilitating DNA end resection.

70 citations

humanDNA methylation

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Molecular BiologybioRxivPreprintMar 23, 2018

RAD51 paralogs regulate double strand break repair pathway choice by limiting Ku complex retention

Fernando Mejías-Navarro, Daniel Gómez-Cabello, Pablo Huertas

This study identifies a new role for RAD51 paralogs in regulating double strand break repair pathway choice by limiting Ku complex retention, favoring homology-mediated repair over non-homologous end-joining.

DOI 10.1101/282855

experimental assay

𝕏 PostBluesky