Author

Victoria Prieto-Echagüe

Institut Pasteur de MontevideoORCID 0000-0001-9924-9591Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Organism:mouse· 1

Frequent Co-Authors

Carlos EscandeiD· 2 shared José Luis BadanoiD· 2 shared Corrine SmoleniD· 1 shared Florencia Irigoı́niD· 1 shared Geraldine SchlappiD· 1 shared Karina Hernández· 1 shared Leonardo SantosiD· 1 shared Lucía Guggeri· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersCell Biology

Carlos EscandeORCID

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2 papers

Recent·Most cited

FSTL1 is an antagonist of ERK1/2 phosphorylation during ciliogenesis and preadipocyte differentiation.

Leonardo Santos, Lucía Guggeri, Carlos Escande, José Luis Badano, Victoria Prieto-Echagüe

This study identifies ERK1/2 phosphorylation as a mediator of FSTL1's role in ciliogenesis and preadipocyte differentiation, showing FSTL1 antagonizes BMP non-canonical signaling.

bioRxiv · Feb 21, 2024 · PreprintCell Biology

Generation and characterization of Ccdc28b mutant mice links the Bardet-Biedl associated gene with social behavioral phenotypes

Matías Fabregat, Sofía Niño, Sabrina Pose, Magdalena Cárdenas-Rodríguez, Corrine Smolen, Mariana Bresque, Karina Hernández, Victoria Prieto-Echagüe, Geraldine Schlapp, Martina Crispo, Patricia Lagos, Natalia Lago, Santhosh Girirajan, Carlos Escande, Florencia Irigoı́n, José Luis Badano

This paper generates Ccdc28b mutant mice and finds they exhibit social behavioral defects and autism-like features, suggesting a link between CCDC28B and autism spectrum disorder beyond its role as a modifier of Bardet-Biedl syndrome.

bioRxiv · Oct 21, 2021 · PreprintGenetics