Author

Benjamin Chun‐Kit Tong

Max Planck Institute for Biological IntelligenceORCID 0000-0002-0112-9274Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Assay:electrophysiology· 1 Approach:experimental assay· 1 Assay:imaging· 1 Organism:mouse· 1 Research Focus:synaptic & neural circuits· 1

Frequent Co-Authors

Angelika Bettina HarbaueriD· 1 shared Ariane Metzger· 1 shared Inmaculada SeguraiD· 1 shared Markus RehmiD· 1 shared Melanie BoerriesiD· 1 shared Simone WanderoyiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

6 top collaborators · 15 mutual co-author links

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1 shared paperNeuroscience

Angelika Bettina HarbauerORCID

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1 paper

Recent·Most cited

A physiological Na+ signal activates mitochondrial PINK1 to couple synaptic depression to caspase-3-dependent spine pruning

Benjamin Chun‐Kit Tong, Inmaculada Segura, Simone Wanderoy, Markus Rehm, Ariane Metzger, Melanie Boerries, Angelika Bettina Harbauer

This study identifies a Na+-PINK1 signaling pathway that links NMDA receptor activity to mitochondrial fission and caspase-3-dependent synapse elimination during long-term depression.

bioRxiv · May 31, 2026 · PreprintNeuroscience