Author

Guangwen Wang

Stanford MedicineORCID 0000-0002-0121-5439Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Systems Biology· 2 Genomics· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 3 Organism:human· 3 Approach:statistical model· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:proteomics· 2 Research Focus:clustering· 1

Frequent Co-Authors

Michael P. SnyderiD· 3 shared Rene Hoffman· 2 shared Songjie CheniD· 2 shared Xun Cheng· 2 shared Benedikt RauscheriD· 1 shared Bettina HaaseiD· 1 shared Daniel HornburgiD· 1 shared Emma MonteiD· 1 shared

Activity by Year

2021-2024 · peak 2/yr

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10 top collaborators · 27 mutual co-author links

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3 shared papersSystems Biology

Michael P. SnyderORCID

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3 papers

Recent·Most cited

GenomicsbioRxivPreprintMay 10, 2024

Personalized transcriptome signatures in a cardiomyopathy stem cell biobank

Emma Monte, Takaaki Furihata, Guangwen Wang…·Michael P. Snyder

The paper establishes a cardiomyopathy stem cell biobank and identifies transcriptomic subtypes and drug response in iPSC-derived cardiomyocytes.

2 citations

humanbulk RNA-seq

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Systems BiologyThe Journal of BiochemistryPeer-reviewedDec 6, 2021

Patient-derived gene and protein expression signatures of NGLY1 deficiency

Benedikt Rauscher, William F. Mueller, Sandra Clauder‐Münster…·Lars Michael Steinmetz

The paper characterizes gene and protein expression changes in NGLY1 deficiency using patient-derived cells and provides a web resource for the data.

13 citations

humanbulk RNA-seq

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Systems BiologybioRxivPreprintMay 28, 2021

Integration and comparison of multi-omics profiles of NGLY1 deficiency plasma and cellular models to identify clinically relevant molecular phenotypes

Songjie Chen, Guangwen Wang, Xiaotao Shen…·Michael P. Snyder

This study integrates metabolomics and proteomics data from NGLY1 deficiency patient samples and cellular models to identify molecular phenotypes linked to immune activation and metabolic disturbances.

4 citations

humanmetabolomics

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