Danielle M. Robertson

Pseudomonas aeruginosa impairs mitochondrial function and metabolism during infection of corneal epithelial cells

This study shows that Pseudomonas aeruginosa infection causes mitochondrial fission and metabolic changes in corneal epithelial cells, which are reversed after antibiotic treatment.

Hyperosmolar stress promotes the release of small extracellular vesicles containing metabolic proteins from corneal epithelial cells

This study shows that hyperosmolar stress increases the release of small extracellular vesicles containing metabolic proteins from corneal epithelial cells, suggesting a potential early indicator of dry eye disease.

IL-1b-mediated Immunometabolic Adaptation in Corneal Epithelial Cells

This study examines how IL-1β affects mitochondrial function in corneal epithelial cells, finding that prolonged exposure leads to metabolic rewiring and increased mitochondrial capacity.

Pseudomonas aeruginosa infection increases palmitoyl carnitine release by host-derived extracellular vesicles

This study identifies palmitoyl carnitine (PAMC) as a metabolite upregulated in host-derived extracellular vesicles during Pseudomonas aeruginosa infection, which modulates immune responses and bacterial growth.