Katharina Brosch

The paper identifies neural correlates of interpersonal resilience, finding larger gray matter volume in the left supramarginal gyrus in individuals with secure attachment despite childhood maltreatment.

This study examines how stressful life events affect adolescent neuroanatomy using ABCD data, finding mostly non-significant associations but some sex-specific effects on cortical thickness and surface area.

The paper investigates how neuroanatomical variability relates to impulsivity in youth using data from the ABCD Study, finding sex-specific and dynamic associations across multiple brain features.

This study investigates the replicability of gray matter correlates of childhood maltreatment across three cohorts and finds no significant replication after controlling for depression.

The paper uses machine learning to link socioeconomic status, scarcity, and neighborhood threat to cortical thickness in youth, finding consistent neural signatures in memory and visual processing regions.

This study uses normative modeling to analyze cortical thickness deviations in MDD patients and healthy controls, finding large overlaps and significant heterogeneity in extreme deviations across individuals.

The paper identifies neural markers of impending initial major depressive episodes by comparing converters, MDD patients, and healthy controls using voxel-wise gray matter volume analysis.

This study investigates how structural brain alterations in schizophrenia relate to network architecture, finding hub susceptibility and shared epicenters across disease stages and disorders.

This study examines how age and parenthood affect whole-brain controllability, finding that parenthood preserves controllability in mothers but not in men.

This paper investigates how cross-validation affects effect size estimation in brain-wide association studies and finds that cross-validation improves generalizability, especially in small samples.

The paper demonstrates a data harmonization process for memory testing across 53 studies, reducing inter-site variance while preserving covariate effects.

This study identifies miR-499-5p as a regulator of neuroplasticity in bipolar disorder by targeting CACNB2, leading to impaired dendritogenesis and cognitive function.