Author

Hazel T. Salunga

Gladstone InstitutesORCID 0000-0002-0582-7352Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1 Research Focus:variant calling· 1

Frequent Co-Authors

Casey A. GiffordiD· 2 shared Deepak SrivastavaiD· 2 shared Sanjeev S. RanadeiD· 2 shared Yu HuangiD· 2 shared Yvanka de SoysaiD· 2 shared Amelia Schricker· 1 shared Antonio del SoliD· 1 shared Aryé Elfenbein· 1 shared

Activity by Year

2018-2019 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersDevelopmental Biology

Casey A. GiffordORCID

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2 papers

Recent·Most cited

Single-cell transcriptome analysis during cardiogenesis reveals basis for organ level developmental anomalies

Yvanka de Soysa, Sanjeev S. Ranade, Satoshi Okawa, Srikanth Ravichandran, Yu Huang, Hazel T. Salunga, Amelia Schricker, Antonio del Sol, Casey A. Gifford, Deepak Srivastava

The paper uses single-cell RNA sequencing to analyze cardiac progenitor cells during normal and abnormal cardiogenesis, identifying transcriptional changes linked to congenital heart defects.

Nature · Jul 24, 2019Peer-reviewedDevelopmental Biology

Oligogenic inheritance of congenital heart disease involving a NKX2-5 modifier

Casey A. Gifford, Sanjeev S. Ranade, Ryan Samarakoon, Hazel T. Salunga, Yvanka de Soysa, Yu Huang, Ping Zhou, Aryé Elfenbein, Stacia K. Wyman, Yen Kim Bui, Kimberly R. Cordes Metzler, Philip C. Ursell, Kathryn N. Ivey, Deepak Srivastava

The paper shows that a congenital heart defect can result from three inherited heterozygous missense variants in MYH7, MKL2, and NKX2-5.

bioRxiv · Feb 20, 2018 · PreprintGenetics