Valery A. Matarazzo

This paper identifies a hypothalamus-brainstem circuit mediated by oxytocin that enhances respiratory sinus arrhythmia during calming behaviors.

The paper shows that cool thermal processing emerges in the neonatal somatosensory cortex and is disrupted in a mouse model of autism.

This study shows that oxytocin administration in neonatal Magel2-deficient mice restores hippocampal circuitry and social memory, suggesting therapeutic potential for autism.

This study identifies early life thermosensory impairments in Magel2-deficient mice and shows oxytocin treatment can rescue these deficits.