Author

Ai Vu Hong

InsermORCID 0000-0002-0872-4295Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Organism:mouse· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:human· 1

Frequent Co-Authors

David IsraeliiD· 2 shared Isabelle RichardiD· 2 shared Abbass JaberiD· 1 shared Adeline Miranda· 1 shared Daniel StockholmiD· 1 shared Elise LachiveriD· 1 shared Fatima Amor· 1 shared Guillaume CorreiD· 1 shared

Activity by Year

2021-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersPhysiology

David IsraeliORCID

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2 papers

Recent·Most cited

Targeting lysosomal damage is a new therapeutic perspective for Duchenne Muscular Dystrophy

Abbass Jaber, Laura Palmieri, Rania Bakour, Elise Lachiver, Ai Vu Hong, Nathalie Bourg, Jérôme Poupiot, Sonia Albini, Daniel Stockholm, Laetitia van Wittenberghe, Adeline Miranda, Nathalie Danièle, Inès Barthélémy, Stéphane Blot, Mai Thao Bui, , David Israeli

This study identifies lysosomal damage as a novel mechanism in Duchenne Muscular Dystrophy and shows that combining trehalose with gene therapy improves outcomes.

bioRxiv · Feb 11, 2025 · PreprintPhysiology

Cholesterol metabolism is a potential therapeutic target in Duchenne Muscular Dystrophy

Fatima Amor, Ai Vu Hong, Guillaume Corre, Mathilde Sanson, Laurence Suel, S. Blaie, Laurent Servais, Thomas Voït, Isabelle Richard, David Israeli

This study identifies dysregulated miRNAs in DMD patients and mice, linking cholesterol metabolism and the mevalonate pathway to disease progression, with Simvastatin treatment showing therapeutic potential.

Journal of Cachexia Sarcopenia and Muscle · May 26, 2021Peer-reviewedGenetics