Author

Katherine E. Schwetye

Washington University in St. LouisORCID 0000-0002-0885-2764Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Immunology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:cell type annotation· 1 Assay:CITE-seq· 1 Research Focus:clustering· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:proteomics· 1

Frequent Co-Authors

Amna Ramadan· 1 shared Andres Bur· 1 shared Ann Marie EgloffiD· 1 shared Anthony J. ApicelliiD· 1 shared Benjamin M. WahleiD· 1 shared Brendan J. KnappiD· 1 shared Chihiro SatoiD· 1 shared Christine AuberleiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperImmunology

Amna Ramadan

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2 papers

Recent·Most cited

High-avidity TCR signaling drives an NKG2A-positive CD8 T cell exhaustion state in human tumors

Riley D. Z. Mullins, Jesse Meir Zaretsky, Thomas F. Barrett, Emily Stoller, Porter Bischoff, Ann Marie Egloff, Cody Huffman, Salma Ramadan, Amna Ramadan, Jessica C. Ley, Katherine E. Schwetye, D K Miller, Jennifer De Los Santos, Anthony J. Apicelli, Nikhil Rammohan, , Ravindra Uppaluri

This study identifies high-avidity TCR signaling via NFAT as a key driver of NKG2A-positive CD8 T cell exhaustion in human tumors, with implications for immunotherapy.

bioRxiv · Aug 27, 2026 · PreprintImmunology

Post-translational modifications distinguish amyloid-β isoform patterns extracted from vascular deposits and parenchymal plaques

Srinivas Koutarapu, Kaleigh F. Roberts, Reid A. Coyle, Jogender Mehla, Chihiro Sato, Gregory Joseph Zipfel, Randall John Bateman, Katherine E. Schwetye, Soumya Mukherjee

The paper identifies differences in post-translational modifications of amyloid-β isoforms between vascular deposits and parenchymal plaques in Alzheimer's disease.

bioRxiv · Jun 11, 2025 · PreprintNeuroscience