Author

Clémence Claussin

Memorial Sloan Kettering Cancer CenterORCID 0000-0002-0994-3596Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Assay:long-read sequencing· 1 Approach:statistical model· 1 Organism:yeast· 1

Frequent Co-Authors

David PorubskỳiD· 1 shared Diana C.J. SpieringsiD· 1 shared Iestyn WhitehouseiD· 1 shared Jacob VazqueziD· 1 shared Michael ChangiD· 1 shared Nancy Halsema· 1 shared Peter M. LansdorpiD· 1 shared Stefan RentasiD· 1 shared

Activity by Year

2017-2022 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 22 mutual co-author links

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1 shared paperMolecular Biology

David PorubskỳORCID

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2 papers

Recent·Most cited

Single-molecule mapping of replisome progression

Clémence Claussin, Jacob Vazquez, Iestyn Whitehouse

The paper develops Replicon-seq to map replisome positions and finds sister replisomes progress consistently but function autonomously, with Rrm3 aiding in overcoming obstacles and termination.

Molecular Cell · Mar 2, 2022Peer-reviewedMolecular Biology

Double-strand breaks are not the main cause of spontaneous sister chromatid exchange in wild-type yeast cells

Clémence Claussin, David Porubskỳ, Diana C.J. Spierings, Nancy Halsema, Stefan Rentas, Victor Guryev, Peter M. Lansdorp, Michael Chang

This study uses Strand-seq to show that most spontaneous sister chromatid exchanges in yeast are not caused by DNA double-strand breaks.

eLife · Dec 12, 2017Peer-reviewedMolecular Biology