Author

Ernesto G. Miranda-Morales

The University of Texas Medical Branch at GalvestonORCID 0000-0002-1129-1750Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Jia Yi LiewiD· 2 shared Ahmet E. Balci· 1 shared Angelo GaitasiD· 1 shared Balaji KrishnaniD· 1 shared Bin GongiD· 1 shared Jiani Bei· 1 shared Madison Samples· 1 shared Mauro MontalbanoiD· 1 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersNeuroscience

Jia Yi LiewORCID

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintSep 9, 2025

Disease-specific tau polymorphs define unique protein interaction networks across proteinopathies

Nicha Puangmalai, Nemil N. Bhatt, Nopparat Suthprasertporn…·Rakez Kayed

The paper identifies disease-specific tau polymorphs and their unique protein interaction networks in Alzheimer's, PSP, and DLB, revealing distinct molecular signatures and therapeutic targets.

DOI 10.1101/2025.09.09.675096

humanproteomics

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NeurosciencebioRxivPreprintApr 3, 2023

Circulating exosomes from Alzheimer's disease suppress VE-cadherin expression and induce barrier dysfunction in recipient brain microvascular endothelial cell

Jiani Bei, Ernesto G. Miranda-Morales, Qini Gan…·Bin Gong

This study shows that Alzheimer's disease circulating exosomes reduce VE-cadherin expression and impair brain endothelial barrier function through RNA cargo-dependent mechanisms.

3 citations

humanexperimental assay

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