Author

Maxime Sartori

Centre National de la Recherche ScientifiqueORCID 0000-0002-1192-1636Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:imaging· 1

Frequent Co-Authors

Damien MaréchaliD· 2 shared Jocelyn LaporteiD· 2 shared Yann HéraultiD· 2 shared A McKenzie· 1 shared Adrien HerlédaniD· 1 shared Alessia LasorsaiD· 1 shared Alexandra WinkeleriD· 1 shared Amandine Flaig· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersNeuroscience

Damien MaréchalORCID

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2 papers

Recent·Most cited

BIN1 genetic risk factor for Alzheimer is sufficient to induce early structural tract alterations in entorhinal cortex-dentate gyrus pathway and related hippocampal multi-scale impairments

Rachel Daudin, Damien Maréchal, R Golgolab, Q Wang, Y Abe, T. Tsurugizawa, Nathalie Bourg, Maxime Sartori, Yann Loe-Mie, Joanna Lipecka, Ida Chiara Guerrera, A McKenzie, Brigitte Potier, P. Dutar, Julia Viard, , Michel Simonneau

This paper shows that BIN1 overexpression in mice leads to early structural and functional impairments in hippocampal pathways associated with Alzheimer's disease, independent of A-beta changes.

bioRxiv · Mar 9, 2021 · PreprintNeuroscience

BIN1 recovers tauopathy-induced long-term memory deficits in mice and interacts with Tau through Thr348 phosphorylation

Maxime Sartori, Tiago Mendes, Shruti Desai, Alessia Lasorsa, Adrien Herlédan, Nicolas Malmanche, Petra M. Mäkinen, Mikael Marttinen, Idir Malki, Julien Chapuis, Amandine Flaig, Anaïs-Camille Vreulx, Marion Ciancia, Philippe Amouyel, Florence Leroux, , Jean‐Charles Lambert

This study shows BIN1 overexpression rescues long-term memory deficits in Tau-overexpressing mice and identifies Thr348 phosphorylation as a key regulator of BIN1-Tau interaction.

Acta Neuropathologica · May 7, 2019Peer-reviewedNeuroscience