Jeffrey C. Magee

This paper develops a model of continual recognition memory using plateau-gated one-shot plasticity, showing it outperforms Hebbian controls in accuracy and memory retention.

This study identifies excessive and ectopic synaptic plasticity in RTT mice as a cause of memory impairment by disrupting stable neural representations.

This study identifies OLM interneurons as providing inhibitory feedback that regulates BTSP and place field formation during spatial learning in mice.

The paper investigates how memory traces influence hippocampal generalization by showing residual activity and synaptic plasticity guide new learning across environments.

The paper shows that dendritic plateau potentials in layer V neurons of the visual cortex rapidly modify network activity during learning, supporting online adaptation.

The paper tracks hippocampal CA1 place cells over 7 days of learning, showing increased stability and task-related representation, supported by synaptic plasticity.

This study shows that hippocampal place cells adapt their reference frames based on experience, switching from allocentric to egocentric in novel environments, with synaptic plasticity playing a key role.

This study shows that CaMKII autophosphorylation is essential for behavioral timescale synaptic plasticity in hippocampal CA1 neurons.

The paper identifies cellular and circuit mechanisms in hippocampal CA3 that underlie attractor dynamics through behavioral timescale synaptic plasticity and entorhinal input.

The paper shows that entorhinal cortex layer 3 activity directs learning-related synaptic plasticity in hippocampal CA1 to create an over-representation of reward locations.

This study shows that bidirectional synaptic plasticity modifies hippocampal place fields by adjusting synaptic weights based on temporal proximity and current synaptic strength.

This study investigates how context-dependent sensory responses in the hippocampus emerge, finding that CA1 neurons inherit conjunctive codes from CA3 inputs.