Author

Silvia Pozzi

Centre de recherche CERVOORCID 0000-0002-1500-8158Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:human· 1 Organism:mouse· 1 Research Focus:protein design· 1 Assay:protein sequence· 1 Assay:protein structure· 1

Frequent Co-Authors

Adriano ChiòiD· 1 shared Alice MigazziiD· 1 shared Andrea CalvoiD· 1 shared Antoine G. GodiniD· 1 shared Ariane Gosselin· 1 shared Bryan J. TraynoriD· 1 shared Cristina MogliaiD· 1 shared Edoardo MicottiiD· 1 shared

Activity by Year

2021-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperNeuroscience

Adriano ChiòORCID

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintFeb 23, 2025

Astrocytic activation of EMMPRIN contributes to their pathological phenotype in ALS.

Gloria Nwamaka Edozie, Jasmine Bélanger, Shanna Pigeyre…·Silvia Pozzi

The paper shows that EMMPRIN activation in astrocytes contributes to their pathological phenotype in ALS and suggests anti-EMMPRIN antibodies as a potential therapeutic approach.

DOI 10.1101/2025.02.23.639749

humanexperimental assay

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NeuroscienceBrainPeer-reviewedSep 14, 2021

Defective cyclophilin A induces TDP-43 proteinopathy: implications for amyotrophic lateral sclerosis and frontotemporal dementia

Laura Pasetto, Maurizio Grassano, Silvia Pozzi…·Valentina Bonetto

This study shows that defective cyclophilin A (PPIA) leads to TDP-43 proteinopathy, contributing to ALS and FTD by disrupting TDP-43 function and causing neurodegeneration.

32 citations

mouseprotein structure

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