Author

Dávid Brenner

Universität UlmORCID 0000-0002-1535-3146Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:scRNA-seq· 1 Research Focus:variant calling· 1 Assay:whole-exome sequencing· 1

Frequent Co-Authors

Aaron HeldiD· 1 shared Alberto CataneseiD· 1 shared Andrea SalzingeriD· 1 shared Andreas HermanniD· 1 shared Annekathrin Rödiger· 1 shared Anže Karlek· 1 shared Axel FreischmidtiD· 1 shared Banaja P. DashiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperNeuroscience

Aaron HeldORCID

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2 papers

Recent·Most cited

NeuroscienceAdvanced SciencePeer-reviewedJan 28, 2026

RIG-I mediated neuron-specific IFN type 1 signaling in FUS-ALS induces neurodegeneration and offers new biomarker-driven individualized treatment options for (FUS-)ALS

Marcel Naumann, Theresa M. Wierschin, Stefanie Kretschmer…·Andreas Hermann

The paper identifies RIG-I-mediated innate immune activation in FUS-ALS spinal motor neurons due to mitochondrial dsRNA accumulation, which can be targeted by JAK-STAT inhibition, offering potential biomarker-driven treatment options.

3 citationstop 2%

humanbulk RNA-seq

GeneticsbioRxivPreprintMar 31, 2025

Somatic mutations in ALS genes in the motor cortex of sporadic ALS patients

Óscar González-Velasco, Rosanna Parlato, Rüstem Yilmaz…·Jochen Hans Weishaupt

This study identifies somatic mutations in ALS genes in the motor cortex of sporadic ALS patients, particularly in excitatory neurons, suggesting a role in disease initiation.

DOI 10.1101/2025.03.31.646375

humanwhole-exome sequencing