Julie S. Haas

This study shows that synaptic GABAA receptor dysfunction in thalamocortical neurons disrupts sleep spindle morphology and impairs fear extinction in mice.

The paper models thalamocortical networks to show that electrical coupling in the TRN reduces cortical correlation to sensory inputs, increasing response rates and duration.

This paper introduces opto-δL, a method to identify and quantify electrical synapses, and applies it to map coupled networks in thalamic and cortical tissues.

The paper uses multi-compartmental models to show that electrical synapse asymmetry arises from multiple factors and affects spike timing between coupled neurons.

The paper demonstrates that tonic spiking in one neuron of an electrically coupled pair induces long-term potentiation of electrical synapses, with asymmetry and calcium dependence.

The paper identifies a novel amygdala-TRN pathway that amplifies sound-evoked responses in the auditory cortex through suppression of spontaneous activity.

The paper uses computational models to show electrical synapses influence both subthreshold and spike activity in feedforward circuits during transient inputs.

The paper shows that electrical synapses in the thalamus help shape how principal neurons respond to brief inputs by truncating, delaying, or inhibiting spike trains.