Mary K. Baylies

The paper identifies how BMP signaling regulates myonuclear properties at the Drosophila larval neuromuscular junction, affecting size, ploidy, and gene expression.

This study shows that cofilin regulates postsynaptic actin organization and neuromuscular junction function in a Drosophila nemaline myopathy model, leading to impaired neurotransmission.

This study shows that Drosophila Tropomodulin is essential for actin-dependent processes in myofiber assembly and maintenance, leading to myopathy-related phenotypes when disrupted.

The paper identifies Twinstar as a regulator of muscle development by screening for Twist-interacting proteins in Drosophila.

This study shows that loss of Drosophila Cofilin disrupts sarcomere structure and function, leading to myopathy, and that proteasome activation can delay muscle deterioration.