Author

Ignacy Czajewski

University of DundeeORCID 0000-0002-1661-5806Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:Drosophila· 2 Assay:ATAC-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:clustering· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Andrew T. FerenbachiD· 3 shared Daan M. F. van AalteniD· 3 shared Allan BayatiD· 1 shared Annette SchenckiD· 1 shared Bijayalaxmi SwainiD· 1 shared Christina FengeriD· 1 shared Daniel Mariappa· 1 shared Erik StorkebaumiD· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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3 shared papersGenetics

Andrew T. FerenbachORCID

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3 papers

Recent·Most cited

GeneticseLifePeer-reviewedOct 21, 2024

Rescuable sleep and synaptogenesis phenotypes in a Drosophila model of O-GlcNAc transferase intellectual disability.

Ignacy Czajewski, Bijayalaxmi Swain, Jiawei Xu…·Daan M. F. van Aalten

This study identifies synaptogenesis and sleep stability defects in a Drosophila model of OGT intellectual disability and shows partial rescue by modulating OGA activity.

11 citations

Drosophilaexperimental assay

𝕏 PostBluesky

Molecular BiologybioRxivPreprintMar 13, 2023

An O-GlcNAc transferase pathogenic variant that affects pluripotent stem cell self-renewal

Michaela Omelková, Christina Fenger, Marta Murray…·Daan M. F. van Aalten

The paper shows that an OGT variant causes reduced pluripotent stem cell self-renewal and lower stem cell marker levels in mouse embryonic stem cells.

3 citations

mouseATAC-seq

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GeneticsbioRxivPreprintJan 10, 2022

Intellectual disability-associated disruption of O-GlcNAcylation impairs neuronal development and cognitive function in Drosophila

Michaela Fencková, Villő Muha, Daniel Mariappa…·Daan M. F. van Aalten

This study shows that OGT mutations impair cognitive function in Drosophila by disrupting O-GlcNAcylation, and that blocking OGA can correct the deficit.

1 citation

Drosophilaexperimental assay

𝕏 PostBluesky