Tony Gutschner

The paper analyzes codon usage patterns in pancreatic cancer to show how oncogenic gene expression is systematically optimized through GC content, preferred codons, and cost-effective amino acids.

This study identifies FOXM1 as a gene regulated by platelets that controls anoikis resistance in pancreatic cancer cells, suggesting it as a potential therapeutic target.

This study shows RNA-seq-derived sequence variations can reliably identify cell lines and detect cross-contamination using machine learning.

This study identifies NEAT1 as a regulator of genome stability through m6A methylation-dependent control of CHD4 in response to DNA damage.

This paper investigates RAVER1's role in controlling alternative splicing and its impact on miR/RISC activity and EMT in cancer cells.