Author

Steven R. Blanke

University of Illinois Urbana-ChampaignORCID 0000-0002-2164-875XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Batcha Tamilselvam· 1 shared D’Feau J. LieuiD· 1 shared Eunji JeongiD· 1 shared Hanlin Mok· 1 shared Heidi Phillips· 1 shared Henry CheniD· 1 shared Ik-Jung KimiD· 1 shared Jordan R. Kryza· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCell Biology

Batcha Tamilselvam

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1 paper

Recent·Most cited

Autophagy suppression in DNA damaged cells occurs through a newly identified p53-proteasome-LC3 axis

D’Feau J. Lieu, Molly K. Crowder, Jordan R. Kryza, Batcha Tamilselvam, Paul J. Kaminski, Ik-Jung Kim, Yingxing Li, Eunji Jeong, Michidmaa Enkhbaatar, Henry Chen, Sophia B. Son, Hanlin Mok, Kenneth A. Bradley, Heidi Phillips, Steven R. Blanke

The paper identifies a p53-proteasome-LC3 axis that suppresses autophagy in DNA damaged cells, linking p53 stabilization to reduced LC3A/B levels.

iScience · Feb 27, 2025Peer-reviewedCell Biology