Author

Molly K. Crowder

Baylor College of MedicineORCID 0000-0001-8962-5231Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:proteomics· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:chromatin & epigenetic regulation· 1 Organism:human· 1

Frequent Co-Authors

Amala K Rougeau· 1 shared Batcha Tamilselvam· 1 shared Charles J. ChoiD· 1 shared D’Feau J. LieuiD· 1 shared Eunji JeongiD· 1 shared Hanlin Mok· 1 shared Heidi Phillips· 1 shared Henry CheniD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCell Biology

Amala K Rougeau

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2 papers

Recent·Most cited

Cell BiologybioRxivPreprintMay 7, 2026

Stress-Responsive Protein IFRD1 Protects Assembled Ribosomes via a Ribosome-Salvaging Mechanism

Charles J. Cho, Molly K. Crowder, Amala K Rougeau…·Jason C. Mills

This study identifies IFRD1 as a ribosome-salvaging factor that stabilizes non-translating 80S ribosomes during ER stress, preventing degradation and cell death.

DOI 10.64898/2026.05.03.720925

mouseproteomics

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Cell BiologyiSciencePeer-reviewedFeb 27, 2025

Autophagy suppression in DNA damaged cells occurs through a newly identified p53-proteasome-LC3 axis

D’Feau J. Lieu, Molly K. Crowder, Jordan R. Kryza…·Steven R. Blanke

The paper identifies a p53-proteasome-LC3 axis that suppresses autophagy in DNA damaged cells, linking p53 stabilization to reduced LC3A/B levels.

2 citations

humanmouse

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