Author

Richard Zapf

Simon Fraser UniversityORCID 0000-0002-2178-8808Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Biochemistry· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:imaging· 1

Frequent Co-Authors

Timothy E. AudasiD· 2 shared Emma Lacroix· 1 shared Evgenia A. Momchilova· 1 shared Isabelle Gracien· 1 shared Krysta Koyle· 1 shared Mythili Padavu· 1 shared Peter J. UnrauiD· 1 shared Ryan D. MoriniD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersBiochemistry

Timothy E. AudasORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Regulation of NFKBIZ by precise Regnase-1 endoribonuclease cleavage and subsequent uridylation

Samira Ebrahimi, Richard Zapf, Isabelle Gracien, Krysta Koyle, Ryan D. Morin, Timothy E. Audas, Peter J. Unrau

The paper shows Regnase-1 cleaves and uridylates NFKBIZ mRNA, enhancing its decay and linking this process to lymphoma and viral pathogenesis.

bioRxiv · Sep 19, 2025 · PreprintBiochemistry

PI3K/AKT Signaling Mediates Stress-Inducible Amyloid Aggregation Through c-myc

Emma Lacroix, Evgenia A. Momchilova, Sahil Chandhok, Mythili Padavu, Richard Zapf, Timothy E. Audas

The paper identifies PI3K/AKT signaling as a mediator of stress-induced amyloid aggregation through c-myc activation and repression of GSK3 degradation.

Cell Reports · Apr 23, 2025Peer-reviewedCell Biology