Karl Foley

This study investigates the role of PP1β in neuronal function by deleting it in mice, revealing its impact on myelination, glutamate release, and action potential propagation.

This study identifies PP1β as a regulator of synaptic plasticity and memory, opposing the role of classic PP1 by promoting spine maturation and LTP.

This study shows that NIPP1 in neural precursor cells regulates tau phosphorylation and myelination, with loss of NIPP1 causing developmental impairments and myelin deficits.

The paper shows that I-2 promotes PP1γ positive regulation of synaptic transmission in hippocampal neurons through enhanced interaction with neurabin.

This study examines the effects of early developmental arsenic exposure on hippocampal synaptic function in mice, finding decreased basal transmission and increased long-term potentiation.