Author

Tomi Tuomainen

University of Eastern FinlandORCID 0000-0002-2387-6369Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:metabolomics· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

Eloi SchmauchiD· 1 shared Johanna KuusistoiD· 1 shared Jussi T. KoivumäkiiD· 1 shared Kyriaki Galani· 1 shared M KellisiD· 1 shared Maike Mona Širinov· 1 shared Marko VendeliniD· 1 shared Mina RabieeiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperPhysiology

Eloi SchmauchORCID

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1 paper

Recent·Most cited

PhysiologybioRxivPreprintMay 15, 2026

Length-dependent hypercontractility decoupled from cellular adaptations is the initial phenotype in MYBPC3-haploinsufficient iPSC-derived engineered cardiac tissues

Tomi Tuomainen, Maike Mona Širinov, Mina Rabiee…·Pasi Tavi

This study identifies length-dependent hypercontractility as the initial phenotype of MYBPC3-haploinsufficiency in iPSC-derived cardiac tissues, decoupled from early transcriptional and metabolic changes.

DOI 10.64898/2026.05.13.724801

humanscRNA-seq

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