Author

Dakota J. Nuttall

University of Wisconsin–MadisonORCID 0000-0002-2472-528XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 2

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Assay:proteomics· 2 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Approach:statistical model· 1

Frequent Co-Authors

Ahmed I. MahmoudiD· 2 shared Jiyoung Bae· 2 shared Timothy J. AballoiD· 2 shared Wyatt G. PaltzeriD· 2 shared Ying GeiD· 2 shared Andrew J. PerciaccanteiD· 1 shared Cassidy Perry· 1 shared Corey G. K. FlynniD· 1 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersDevelopmental Biology

Ahmed I. MahmoudORCID

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2 papers

Recent·Most cited

Integrated Proteomics Identifies Troponin I Isoform Switch as a Regulator of a Sarcomere-Metabolism Axis During Cardiac Regeneration

Timothy J. Aballo, Jiyoung Bae, Wyatt G. Paltzer, Emily A. Chapman, Andrew J. Perciaccante, Melissa R. Pergande, Rebecca J. Salamon, Dakota J. Nuttall, Morgan W. Mann, Ying Ge, Ahmed I. Mahmoud

The paper identifies ssTnI overexpression as enhancing cardiomyocyte proliferation and regenerative capacity post-MI through metabolic shifts.

Cardiovascular Research · Apr 17, 2025Peer-reviewedDevelopmental Biology

mTORC1 Regulates the Metabolic Switch of Postnatal Cardiomyocytes During Regeneration

Wyatt G. Paltzer, Timothy J. Aballo, Jiyoung Bae, Corey G. K. Flynn, Kayla N. Wanless, Katharine A. Hubert, Dakota J. Nuttall, Cassidy Perry, Raya Nahlawi, Ying Ge, Ahmed I. Mahmoud

This study shows that mTORC1 inhibition accelerates the metabolic switch from glycolysis to fatty acid oxidation in neonatal heart injury, impairing regeneration.

bioRxiv · Sep 12, 2023Developmental Biology