Author

Emil Aagaard Thomsen

Aarhus UniversityORCID 0000-0002-2685-7745Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Approach:machine learning (classical)· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

Anton MarkoviD· 1 shared Diana SharyshiD· 1 shared Jacob Fog BentzoniD· 1 shared Jacob Giehm MikkelseniD· 1 shared Julián Albarrán-JuáreziD· 1 shared Laura Alonso-HerranziD· 1 shared Lise Filt JenseniD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

7 top collaborators · 21 mutual co-author links

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1 shared paperCell Biology

Anton MarkovORCID

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1 paper

Recent·Most cited

Loss of physiological stretch elicits an inflammatory, plaque-like transcriptional state in cultured human aortic smooth muscle cells

Lise Filt Jensen, Anton Markov, Laura Alonso-Herranz, Diana Sharysh, Emil Aagaard Thomsen, Jacob Giehm Mikkelsen, Jacob Fog Bentzon, Julián Albarrán-Juárez

This study shows that physiological stretch reduces inflammation in cultured human aortic smooth muscle cells, while static culture or excessive stretch promotes an inflammatory, plaque-like state.

bioRxiv · Oct 29, 2025 · PreprintCell Biology