Author

Fabio Iannelli

European Institute of OncologyORCID 0000-0002-2777-1417Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2 Cell Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:cell-cell communication· 2 Research Focus:differential expression· 2 Assay:DNA methylation· 2 Approach:statistical model· 2 Research Focus:cancer & tumor microenvironment· 1 Assay:ChIP-seq· 1

Frequent Co-Authors

Fabrizio d’Adda di FagagnaiD· 2 shared Matteo CabriniiD· 2 shared Roberto ChiarleiD· 2 shared Sofia FranciaiD· 2 shared Adelaide Riccardi· 1 shared Alessandro Galbiati· 1 shared Alessia di LilloiD· 1 shared Alessio ColantoniiD· 1 shared

Activity by Year

2018-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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Curved chords = mutual co-authorship

2 shared papersMolecular Biology

Fabrizio d’Adda di FagagnaORCID

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Cancer BiologyCell Reports MedicinePeer-reviewedMar 31, 2026

Epstein-Barr Virus Orchestrates Spatial Reorganization and Immunomodulation within the Classic Hodgkin Lymphoma Tumor Microenvironment

Yao Yu Yeo, Huaying Qiu, Yunhao Bai…·Sizun Jiang

This study uses spatial multi-omics to compare EBV-positive and EBV-negative cHL, identifying distinct TME features and immunological signatures linked to EBV.

3 citations

humanspatial transcriptomics

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Molecular BiologyCell ReportsPeer-reviewedDec 1, 2025

DROSHA, DICER and Damage-Induced long ncRNA control BMI1-dependent transcriptional repression at DNA double-strand break

Francesca Esposito, Ilaria Capozzo, Adelaide Riccardi…·Sofia Francia

The paper shows that dilncRNA, DROSHA, and DICER control DISC by recruiting BMI1 and promoting H2A-K119 ubiquitination at DNA double-strand breaks.

8 citations

humanDNA methylation

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Cancer BiologyLeukemiaPeer-reviewedApr 2, 2025

HDAC1 acts as tumor suppressor in ALK-positive anaplastic large-cell lymphoma: Implications for HDAC inhibitor therapy

Maša Zrimšek, Kristina Draganić, Anna Malzer…·Gerda Egger

This study shows HDAC1 suppresses ALK-positive lymphoma growth, but systemic HDAC inhibition can still be therapeutic.

8 citations

mouseexperimental assay

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Cell BiologybioRxivPreprintFeb 7, 2018

DROSHA associates to DNA damage sites and is required for DNA repair

Matteo Cabrini, Marco Roncador, Alessandro Galbiati…·Sofia Francia

This study shows DROSHA localizes to DNA damage sites and is essential for NHEJ DNA repair.

DOI 10.1101/261289

humanDNA methylation

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