Steve M. Taylor

The paper identifies superinfection as a key mechanism in sustaining complex Plasmodium falciparum infections in mosquitoes by analyzing genetic diversity in a longitudinal cohort.

The paper investigates the association between host sickle cell haemoglobin and parasite genetic variation in malaria severity, finding that parasite alleles linked to HbS show strong linkage disequilibrium and frequency covariation with HbS.

This study identifies transcriptomic changes in Plasmodium falciparum in HbAS red blood cells, showing altered protein export and stress response mechanisms that may contribute to malaria protection.