Author

Jonathan J. Quan

University of California, DavisORCID 0000-0002-3312-6908Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell-cell communication· 1 Assay:imaging· 1 Assay:proteomics· 1

Frequent Co-Authors

Adrian SheehyiD· 1 shared Arash BellafardiD· 1 shared Avaneesh Murugesan· 1 shared Daniel Benjamin AharoniiD· 1 shared Janet Nuno· 1 shared Jorge E. ContrerasiD· 1 shared Manuel F. MuñoziD· 1 shared Mauricio A. LilloiD· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperPhysiology

Adrian SheehyORCID

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2 papers

Recent·Most cited

Impaired S-nitrosylation of Cx43 prevents arrhythmogenicity and myocardial injury upon cardiac stress in Duchenne Muscular Dystrophy

Manuel F. Muñoz, Pablo S. Gaete, Jonathan J. Quan, Thao T Nguyen, Janet Nuno, Adrian Sheehy, Priscila Ágape Pacheco Pereira Araújo, Pía C. Burboa, Mauricio A. Lillo, Jorge E. Contreras

This study shows that reducing S-nitrosylation of Cx43 in DMD mice prevents arrhythmias and myocardial injury under cardiac stress.

Cardiovascular Research · Aug 7, 2026Peer-reviewedPhysiology

Accelerated social representational drift in the nucleus accumbens in a model of autism

Pingping Zhao, Xing Chen, Arash Bellafard, Avaneesh Murugesan, Jonathan J. Quan, Daniel Benjamin Aharoni, Peyman Golshani

This study identifies a stable population code for social interaction in the nucleus accumbens, which is degraded in an ASD mouse model and can be modulated by optogenetic inhibition.

bioRxiv · Aug 5, 2023 · PreprintNeuroscience