Author

Abigail H. Cleveland

University of North Carolina at Chapel HillORCID 0000-0002-3502-5940Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

Timothy R. GershoniD· 2 shared Alejandra I. Romero-MoralesiD· 1 shared Claudia Rodriguez· 1 shared Daniel Shiloh MalawskyiD· 1 shared Frances Reed· 1 shared Ian J. DavisiD· 1 shared José E. Velázquez VegaiD· 1 shared Laurent Alfonso Azcona· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersNeuroscience

Timothy R. GershonORCID

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2 papers

Recent·Most cited

PRC2 disruption in cerebellar progenitors produces cerebellar hypoplasia and aberrant myoid differentiation without blocking medulloblastoma growth

Abigail H. Cleveland, Daniel Shiloh Malawsky, Mehal Churiwal, Claudia Rodriguez, Frances Reed, Matthew Schniederjan, José E. Velázquez Vega, Ian J. Davis, Timothy R. Gershon

This study shows that PRC2 maintains neural identity in cerebellar progenitors and medulloblastoma, but its disruption does not block medulloblastoma progression.

bioRxiv · Jul 11, 2022 · PreprintNeuroscience

Oligodendrocytes depend on MCL-1 to prevent spontaneous apoptosis and white matter degeneration

Abigail H. Cleveland, Alejandra I. Romero-Morales, Laurent Alfonso Azcona, Melisa Herrero, Viktoriya D. Nikolova, Sheryl S. Moy, Orna Elroy‐Stein, Vivian Gama, Timothy R. Gershon

This study shows that oligodendrocytes require MCL-1 to prevent apoptosis and white matter degeneration, suggesting MCL-1 deficiency contributes to leukodystrophy.

Cell Death and Disease · Dec 6, 2021Peer-reviewedNeuroscience