Author

Weijing Tang

Stanford MedicineORCID 0000-0002-3543-9924Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 3 Approach:statistical model· 3 Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Research Focus:cell type annotation· 1 Organism:Drosophila· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1

Frequent Co-Authors

Inma CobosiD· 3 shared Marcos Otero‐GarcíaiD· 3 shared Jie PaniD· 2 shared Alan Edward RentoniD· 1 shared Alicia Casella· 1 shared Alison GoateiD· 1 shared Andrew F. TeichiD· 1 shared Angela E. Madira· 1 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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3 shared papersNeuroscience

Inma CobosORCID

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3 papers

Recent·Most cited

NeuroscienceNature CommunicationsPeer-reviewedJan 31, 2026

Molecular Signatures of Resilience to Alzheimer's Disease in Neocortical Layer 4 Neurons

S. Akila Parvathy Dharshini, Jorge Sanz‐Ros, Jie Pan…·Inma Cobos

The paper identifies molecular signatures of resilience in neocortical layer 4 neurons in Alzheimer's disease using single-cell and spatial transcriptomics.

19 citationstop 1%

humanmouse

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NeuroscienceNeuronPeer-reviewedJul 25, 2022

Single-soma transcriptomics of tangle-bearing neurons in Alzheimer's disease reveals the signatures of tau-associated synaptic dysfunction

Marcos Otero‐García, Sameehan Uday Mahajani, Debia Wakhloo…·Inma Cobos

The paper identifies gene expression signatures in Alzheimer's disease neurons with tau aggregates, revealing shared and subtype-specific molecular changes related to synaptic function and metabolism.

262 citationstop 1%

humanbulk RNA-seq

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NeuroscienceActa NeuropathologicaPeer-reviewedOct 31, 2021

Genome-wide association study and functional validation implicates JADE1 in tauopathy

Kurt W. Farrell, SoongHo Kim, Natalia Han…·John F. Crary

This study identifies JADE1 as a novel genetic factor in tauopathy through genome-wide association and functional validation in human and Drosophila models.

35 citationstop 5%

humanDrosophila

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