Author

Rachel Bayley

University of BirminghamORCID 0000-0002-3570-5532Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

Abeer M. ShaabaniD· 1 shared Amber Stiby· 1 shared Anna Munsey· 1 shared Bohdana-Myroslava Briantseva· 1 shared Ciara M. Ward· 1 shared Clare C. DaviesiD· 1 shared Ellie Sweatman· 1 shared Graeme C.M. SmithiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperCancer Biology

Abeer M. ShaabanORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

MYBL2 maintains stemness and promotes theta-mediated end joining in triple negative breast cancer

Rachel Bayley, Anna Munsey, Suman Ahmed, Ciara M. Ward, Amber Stiby, Bohdana-Myroslava Briantseva, Kirendeep Jawanda, Abeer M. Shaaban, Marco Saponaro, Graeme C.M. Smith, Robert B. Clarke, Clare C. Davies, Paloma García

This study shows MYBL2 maintains stemness and promotes theta-mediated end joining in TNBC, and identifies Pol{theta}/ATR inhibition as a therapeutic strategy.

bioRxiv · Apr 15, 2025 · PreprintCancer Biology

SETD1A-dependent EME1 transcription drives PARPi sensitivity in HR deficient tumour cells

Ellie Sweatman, Rachel Bayley, Richad Selemane, Martin R. Higgs

This study shows that SETD1A promotes PARPi sensitivity in HR-deficient tumor cells by regulating EME1 transcription, and its loss leads to resistance.

British Journal of Cancer · Feb 24, 2025Peer-reviewedCancer Biology