Nives Zimmermann

This study identifies PTPN11 mutations as potential initiating events in AML when paired with NPM1 mutations, revealing their co-driver role and immune diversity in patient samples.

This study investigates how eosinophilic inflammation causes heart damage in hypereosinophilia by using a mouse model and single-cell RNA sequencing to identify cell types and pathways involved.

This paper identifies three genetic loci associated with heart disease in a mouse model of eosinophilic myocarditis through linkage analysis of recombinant populations.