Author

Bayard L. Powell

Atrium Health Wake Forest Baptist

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Genetic & Genomic Medicine· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1

Frequent Co-Authors

Andrew J. CarrolliD· 2 shared Ann‐Kathrin EisfeldiD· 2 shared Deedra NicoletiD· 2 shared Jonathan E. KolitziD· 2 shared Krzysztof MrózekiD· 2 shared Linde A. MilesiD· 2 shared Richard Maury StoneiD· 2 shared Robert L. BowmaniD· 2 shared

Activity by Year

2020-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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Curved chords = mutual co-authorship

2 shared papersCancer Biology

Andrew J. CarrollORCID

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Author profile →

3 papers

Recent·Most cited

Prognostic and Therapeutic Implications of BRAF Mutations in Acute Myeloid Leukemia

Darren Lee, Yazan Abu-Shihab, Kate Plas, Deedra Nicolet, Krzysztof Mrózek, Mark J. Routbort, Keyur Pravinchandra Patel, Christopher James Walker, Jill L. Buss, Andrew R. Stiff, Andrea Laganson, Courtney Denton Dinardo, Naval Guastad Daver, Tapan Mahendra Kadia, Farhad Ravandi, , Linde A. Miles

This study identifies BRAF mutations in AML, showing poor outcomes and unique co-mutation patterns, suggesting potential targeted therapies.

Leukemia · Jun 15, 2026Peer-reviewedCancer Biology

PTPN11 Mutation Clonal Hierarchy in Acute Myeloid Leukemia

Sydney Fobare, Chia Sharpe, K. R. Quinn, Kinsey Bryant, Linde A. Miles, Robert L. Bowman, Carolyn M. Cheney, Casie Furby, Marissa Long, Kaytlynn Fyock, Benjamin Wronowski, James R. Lerma, Krzysztof Mrózek, Deedra Nicolet, Thomas M. Sesterhenn, , John C. Byrd

This study identifies PTPN11 mutations as potential initiating events in AML when paired with NPM1 mutations, revealing their co-driver role and immune diversity in patient samples.

JCI Insight · Feb 23, 2026Peer-reviewedCancer Biology

SMARCA4 Mutations in KRAS-mutant Lung Adenocarcinoma: A Multi-cohort Analysis

Liang Liu, Tamjeed Ahmed, William Jeffrey Petty, Stefan C. Grant, Jimmy Ruiz, Thomas William Lycan, Ümit Topaloĝlu, Ping‐Chieh Chou, Lance David Miller, Gregory A. Hawkins, Martha A. Alexander‐Miller, Stacey S. O’Neill, Bayard L. Powell, Ralph B. D’Agostino, Reginald F. Munden, , Wei Zhang

This study analyzes SMARCA4 mutations in KRAS-mutant lung adenocarcinoma across multiple cohorts, finding adverse clinical outcomes associated with SMARCA4 mutations in both non-immunotherapy and immunotherapy-treated patients.

Molecular Oncology · Oct 26, 2020Peer-reviewedGenetic & Genomic Medicine