Author

Magnus Tobiasson

Karolinska University HospitalORCID 0000-0002-3633-5852Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Genomics· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Approach:statistical model· 2 Approach:benchmark study· 1 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Assay:DNA methylation· 1 Research Focus:protein structure prediction· 1 Assay:proteomics· 1

Frequent Co-Authors

Agnes J. VialeiD· 1 shared Akifumi Takaori‐KondoiD· 1 shared Alexandra G. SmithiD· 1 shared Andrea PellagattiiD· 1 shared Anne PaniD· 1 shared Araxe Sarian· 1 shared Arjan A. van de LoosdrechtiD· 1 shared Asif JavediD· 1 shared

Activity by Year

2020-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperGenomics

Agnes J. VialeORCID

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2 papers

Recent·Most cited

Azacitidine Response in Myelodysplastic Syndromes is Marked by NK-like CD8 T-Cell Expansion and CXCL12+ Reticular Cell Remodeling

Henry R. Hampton, Anne Pan, Michael J. Carnell, Bofei Wang, Diana Shinko, Maria A. Kasherman, Iveta Šlapetová, Swapna Joshi, Mary N.T. Nguyen, Feng Yan, Sarah Davidson, Nick F.Y. Choi, Jason W.H. Wong, Nicodemus T. Tedla, Devendra Hiwase, , John E. Pimanda

The paper identifies NK-like CD8 T-cell expansion and CXCL12+ reticular cell remodeling as markers of azacitidine response in MDS through longitudinal bone marrow profiling.

bioRxiv · Jul 8, 2026 · PreprintCancer Biology

Implications of TP53 Allelic State for Genome Stability, Clinical Presentation and Outcomes in Myelodysplastic Syndromes

Elsa Bernard, Yasuhito Nannya, Robert Paul Hasserjian, Sean M. Devlin, Heinz Tuechler, Juan Santiago Medina-Martinez, Tetsuichi Yoshizato, Yusuke Shiozawa, Ryunosuke Saiki, Luca Malcovati, Max Fine Levine, Juan E. Arango, Yangyu Zhou, Francesc Gessé Solé, Catherine A. Cargo, , Elli Papaemmanuil

The paper examines TP53 allelic states in MDS patients, finding that bi-allelic targeting is linked to worse outcomes, while mono-allelic targeting does not differ from wild-type patients.

Nature Medicine · Aug 3, 2020Peer-reviewedGenomics