Author

Francesca Montarolo

Neuroscience Institute Cavalieri OttolenghiORCID 0000-0002-3633-8746Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2 Genetics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3

Frequent Co-Authors

Annalisa BuffoiD· 2 shared Enrica BodaiD· 2 shared Martino BonatoiD· 2 shared Niccolò Di CintioiD· 2 shared Roberta ParolisiiD· 2 shared Alessandro BrussinoiD· 1 shared Alfredo BruscoiD· 1 shared Angelisa FrascaiD· 1 shared

Activity by Year

2018-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersNeuroscience

Annalisa BuffoORCID

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3 papers

Recent·Most cited

Oligodendroglial deletion of the microcephaly gene Cit-k disrupts cortical connectivity and cognitive function

Martino Bonato, Federica Marchiotto, Maryam Khastkhodaei Ardakani, Federico G.P. Ferrari, Niccolò Di Cintio, Annamaria Renna, Ottavia Maria Roggero, Francesca Montarolo, Valentina Cerrato, Angelisa Frasca, Benedetto Sacchetti, Annalisa Buffo, Marco Cambiaghi, Enrica Boda

This study shows that deleting Cit-k in oligodendroglia causes hypomyelination and cognitive deficits in mice, independent of neuronal defects.

bioRxiv · Aug 8, 2026 · PreprintNeuroscience

Acute particulate matter (PM10) exposure selectively triggers behavioral alterations in the presymptomatic Experimental Autoimmune Encephalomyelitis (EAE) mouse model of Multiple Sclerosis

Martino Bonato, Francesca Montarolo, Roberta Parolisi, Maura Caudana, Giuliana Abbadessa, Vita Cardinale, Giulia Nato, Claudio Pandino, Niccolò Di Cintio, Valentina Schiavo, Maurizio Giustetto, Federico Luzzati, Silvia De Francia, Antonio Bertolotto, Annalisa Buffo, Enrica Boda

The paper shows that PM10 exposure affects behavior in presymptomatic EAE mice but not healthy mice, indicating a selective vulnerability in immunologically primed individuals.

European Journal of Neuroscience · Nov 1, 2025Peer-reviewedNeuroscience

Mice harboring a SCA28 patient mutation in AFG3L2 develop late-onset ataxia associated with enhanced mitochondrial proteotoxicity.

Cecilia Mancini, Eriola Hoxha, Luisa Iommarini, Alessandro Brussino, Uwe Richter, Francesca Montarolo, Claudia Cagnoli, Roberta Parolisi, Diana Iulia Gondor Morosini, Valentina Nicolò, Francesca Maltecca, Luísa Muratori, Giulia Ronchi, Stefano Geuna, Francesca Arnaboldi, , Filippo Tempia

This study creates a new mouse model with a patient-derived SCA28 mutation and shows it leads to late-onset ataxia and mitochondrial dysfunction.

Neurobiology of Disease · Oct 30, 2018Peer-reviewedGenetics