Annalisa Buffo

This study validates allele-specific LMNB1 silencing in patient-derived astrocytes as a therapeutic approach for ADLD by showing restored physiological levels and functional improvements.

This study shows that deleting Cit-k in oligodendroglia causes hypomyelination and cognitive deficits in mice, independent of neuronal defects.

The paper shows that brain injury reactivates a developmental program in astrocytes to generate transient LGE-class interneurons that integrate into neural circuits.

This study shows that anodal tDCS promotes myelin repair and enhances oligodendroglial survival and maturation in the injured motor cortex of mice, while also inducing myelin plasticity in the uninjured cortex.

The paper shows that PM10 exposure affects behavior in presymptomatic EAE mice but not healthy mice, indicating a selective vulnerability in immunologically primed individuals.

The paper shows that striatal astrocytes have widespread neurogenic potential that is activated focally after injury, with behavior similar to canonical niches.