Author

Maryam Khastkhodaei Ardakani

Neuroscience Institute Cavalieri OttolenghiORCID 0000-0002-8918-9377Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Annalisa BuffoiD· 2 shared Valentina CerratoiD· 2 shared Alfredo BruscoiD· 1 shared Angelisa FrascaiD· 1 shared Annamaria Renna· 1 shared Arianna Contato· 1 shared Benedetto SacchettiiD· 1 shared Elena SignorinoiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersNeuroscience

Annalisa BuffoORCID

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2 papers

Recent·Most cited

Functional validation of allele-specific LMNB1 silencing in patient-derived astrocytes as a therapeutic option for Autosomal Dominant Leukodystrophy

Martina Lorenzati, Arianna Contato, Fernando Josa‐Prado, Marta Ribodino, Maryam Khastkhodaei Ardakani, Ersilia Nicorvo, Elena Signorino, Giacomo Turrini, Valentina Cerrato, Roberta Parolisi, Filippo Lucchini, Gabriele Piacenti, Giacomo Donati, Valentina Proserpio, Juan Antonio García‐León, , Annalisa Buffo

This study validates allele-specific LMNB1 silencing in patient-derived astrocytes as a therapeutic approach for ADLD by showing restored physiological levels and functional improvements.

bioRxiv · Sep 15, 2026 · PreprintNeuroscience

Oligodendroglial deletion of the microcephaly gene Cit-k disrupts cortical connectivity and cognitive function

Martino Bonato, Federica Marchiotto, Maryam Khastkhodaei Ardakani, Federico G.P. Ferrari, Niccolò Di Cintio, Annamaria Renna, Ottavia Maria Roggero, Francesca Montarolo, Valentina Cerrato, Angelisa Frasca, Benedetto Sacchetti, Annalisa Buffo, Marco Cambiaghi, Enrica Boda

This study shows that deleting Cit-k in oligodendroglia causes hypomyelination and cognitive deficits in mice, independent of neuronal defects.

bioRxiv · Aug 8, 2026 · PreprintNeuroscience